For renal failure patients, carambola may be a poison that has not attracted widespread attention due to its narrow influence on the population.
In recent years, there have been reports of cases of carambola poisoning in patients with chronic kidney disease in the world.
Generally, carambola poisoning can cause intractable hiccup, limb numbness, muscle strength decline, abnormal skin sensation, insomnia, excitement, disorganized thinking, epilepsy, lethargy, coma, diarrhea, hematuria and other poisoning symptoms.
Many doctors have also seen cases of kidney diseases aggravated by eating carambola.
For example, primary nephrotic syndrome is prone to relapse after eating carambola, uremic patients on peritoneal dialysis and hemodialysis are prone to poisoning coma after eating carambola, and even some children have hematuria after eating carambola.
According to experimental studies, this may be related to the irritant neurotoxin contained in carambola.
In addition, animal experiments have also shown that carambola can cause damage to the basement membrane of glomerular capillary, damage to the foot process of epithelial cells, and lead to hematuria. Its mechanism is highly likely to be related to allergy.
We also found that the symptoms of poisoning disappeared rapidly when hemodialysis was used to rescue coma patients with severe poisoning, suggesting that hemodialysis has a good effect on removing toxins from carambola.
It is worth noting that carambola is best not to be eaten by patients with kidney disease or those who have had a history of kidney disease, in order to prevent recurrence of kidney disease or life-threatening poisoning.